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Another approach that has morerecently been explored is to focus on the p53/cofactor interactions that a gain-of-function mutant p53 protein disrupts; blocking such mutant p53 interactions mightregain p53 activity, [6 , 16].

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T2-weighted image showsmore extensive surrounding hyperintense signal abnormalitiesreflecting edema (c). The Diabetes Prevention Project(DPP) study demonstrated that multiple metabolic risk factorscan be controlled and type 2 diabetes prevented or delayed bycontrolling weight with regular exercise. The p53 protein integrates variousstress signals into a growth restrictive cellular response

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Treatment for STEMI is augmented with a supplemental30 mg intravenous injection.

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